Filtros : "ICB" "FISIOLOGIA" "Journal of Endocrinology" Removidos: "HU" "Nunes, Maria Tereza" "Fmrp-Usp" Limpar

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  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, GLUCAGON, ELETROFISIOLOGIA, GLICOSE, ILHOTAS DE LANGERHANS, HIPOGLICEMIA

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      GAO, Rui et al. α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, v. 26, n. 2, p. 1-20, 2023Tradução . . Disponível em: https://doi.org/10.1530/JOE-22-0295. Acesso em: 22 maio 2024.
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      Gao, R., Acreman, S., Ma, J., Wendt, A., Quan Zhang,, & Abdulkader, F. (2023). α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, 26( 2), 1-20. doi:10.1530/JOE-22-0295
    • NLM

      Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0295
    • Vancouver

      Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0295
  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: HISTOLOGIA, FISIOLOGIA

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      LIMA, Maria H. M. et al. Up-regulation of the phosphatidylinositol 3-kinase/protein kinase B pathway in the ovary of rats by chronic treatment with hCG and insulin. Journal of Endocrinology, v. 190, p. 451-459, 2006Tradução . . Disponível em: https://doi.org/10.1677/joe.1.06226. Acesso em: 22 maio 2024.
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      Lima, M. H. M., Souza, L. C., Caperuto, L. C., Bevilacqua, E. M. A. F., Gasparetti, A. L., Zanuto, R., et al. (2006). Up-regulation of the phosphatidylinositol 3-kinase/protein kinase B pathway in the ovary of rats by chronic treatment with hCG and insulin. Journal of Endocrinology, 190, 451-459. doi:10.1677/joe.1.06226
    • NLM

      Lima MHM, Souza LC, Caperuto LC, Bevilacqua EMAF, Gasparetti AL, Zanuto R, Saad MJ, Carvalho CR de O. Up-regulation of the phosphatidylinositol 3-kinase/protein kinase B pathway in the ovary of rats by chronic treatment with hCG and insulin [Internet]. Journal of Endocrinology. 2006 ; 190 451-459.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.06226
    • Vancouver

      Lima MHM, Souza LC, Caperuto LC, Bevilacqua EMAF, Gasparetti AL, Zanuto R, Saad MJ, Carvalho CR de O. Up-regulation of the phosphatidylinositol 3-kinase/protein kinase B pathway in the ovary of rats by chronic treatment with hCG and insulin [Internet]. Journal of Endocrinology. 2006 ; 190 451-459.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.06226
  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA, HORMÔNIO DO CRESCIMENTO, ESTRESSE, ALIMENTOS, GRELINA, OBESIDADE, GRAVIDEZ, HIPOTÁLAMO, RESISTÊNCIA À INSULINA, HOMEOSTASE

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      TAVARES, Mariana Rosolen e FRAZÃO, Renata e DONATO JUNIOR, José. Understanding the role of growth hormone in situations of metabolic stress. Journal of Endocrinology, v. 256, n. 1, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1530/JOE-22-0159. Acesso em: 22 maio 2024.
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      Tavares, M. R., Frazão, R., & Donato Junior, J. (2022). Understanding the role of growth hormone in situations of metabolic stress. Journal of Endocrinology, 256( 1), 1-11. doi:10.1530/JOE-22-0159
    • NLM

      Tavares MR, Frazão R, Donato Junior J. Understanding the role of growth hormone in situations of metabolic stress [Internet]. Journal of Endocrinology. 2022 ; 256( 1): 1-11.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0159
    • Vancouver

      Tavares MR, Frazão R, Donato Junior J. Understanding the role of growth hormone in situations of metabolic stress [Internet]. Journal of Endocrinology. 2022 ; 256( 1): 1-11.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0159
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      GOUVEIA, Cecília Helena de Azevedo et al. Thyroid hormone stimulation of osteocalcin gene expression in ROS 17/2.8 cells is mediated by transcriptional and post-transcriptional mechanism. Journal of Endocrinology, v. 170, p. 667-675, 2001Tradução . . Disponível em: https://doi.org/10.1677/joe.0.1700667. Acesso em: 22 maio 2024.
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      Gouveia, C. H. de A., Schultz, J. J., Bianco, A. C., & Brent, G. A. (2001). Thyroid hormone stimulation of osteocalcin gene expression in ROS 17/2.8 cells is mediated by transcriptional and post-transcriptional mechanism. Journal of Endocrinology, 170, 667-675. doi:10.1677/joe.0.1700667
    • NLM

      Gouveia CH de A, Schultz JJ, Bianco AC, Brent GA. Thyroid hormone stimulation of osteocalcin gene expression in ROS 17/2.8 cells is mediated by transcriptional and post-transcriptional mechanism [Internet]. Journal of Endocrinology. 2001 ; 170 667-675.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1700667
    • Vancouver

      Gouveia CH de A, Schultz JJ, Bianco AC, Brent GA. Thyroid hormone stimulation of osteocalcin gene expression in ROS 17/2.8 cells is mediated by transcriptional and post-transcriptional mechanism [Internet]. Journal of Endocrinology. 2001 ; 170 667-675.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1700667
  • Source: Journal of Endocrinology. Unidades: ICB, EACH

    Subjects: ANATOMIA, HISTOLOGIA, FISIOLOGIA

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      VILLICEV, Cássio M. et al. Thyroid hormone receptor b-specific agonist GC-1 increases energy expenditure and prevents fat-mass accumulation in rats. Journal of Endocrinology, v. 193, n. 1, p. 21-29, 2007Tradução . . Disponível em: https://doi.org/10.1677/joe.1.07066. Acesso em: 22 maio 2024.
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      Villicev, C. M., Freitas, F. R. de S. e, Aoki, M. S., Taffarel, C., Scanlan, T. S., Moriscot, A. S., et al. (2007). Thyroid hormone receptor b-specific agonist GC-1 increases energy expenditure and prevents fat-mass accumulation in rats. Journal of Endocrinology, 193( 1), 21-29. doi:10.1677/joe.1.07066
    • NLM

      Villicev CM, Freitas FR de S e, Aoki MS, Taffarel C, Scanlan TS, Moriscot AS, Ribeiro MO, Bianco AC, Gouveia CH de A. Thyroid hormone receptor b-specific agonist GC-1 increases energy expenditure and prevents fat-mass accumulation in rats [Internet]. Journal of Endocrinology. 2007 ; 193( 1): 21-29.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.07066
    • Vancouver

      Villicev CM, Freitas FR de S e, Aoki MS, Taffarel C, Scanlan TS, Moriscot AS, Ribeiro MO, Bianco AC, Gouveia CH de A. Thyroid hormone receptor b-specific agonist GC-1 increases energy expenditure and prevents fat-mass accumulation in rats [Internet]. Journal of Endocrinology. 2007 ; 193( 1): 21-29.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.07066
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      ANHÊ, Gabriel Forato et al. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology, v. 195, n. 1, p. 17-27, 2007Tradução . . Acesso em: 22 maio 2024.
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      Anhê, G. F., Nogueira, T. C. A., Nicoletti-Carvalho, J. E., Lellis-Santos, C., Barbosa, H. C., Cipolla Neto, J., et al. (2007). Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology, 195( 1), 17-27.
    • NLM

      Anhê GF, Nogueira TCA, Nicoletti-Carvalho JE, Lellis-Santos C, Barbosa HC, Cipolla Neto J, Bosqueiro JR, Boschero AC, Bordin S. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology. 2007 ; 195( 1): 17-27.[citado 2024 maio 22 ]
    • Vancouver

      Anhê GF, Nogueira TCA, Nicoletti-Carvalho JE, Lellis-Santos C, Barbosa HC, Cipolla Neto J, Bosqueiro JR, Boschero AC, Bordin S. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology. 2007 ; 195( 1): 17-27.[citado 2024 maio 22 ]
  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      PEDROSO, João A. B. et al. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance. Journal of Endocrinology, v. 235, n. 3, p. 207-222, 2017Tradução . . Disponível em: https://doi.org/10.1530/JOE-17-0255. Acesso em: 22 maio 2024.
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      Pedroso, J. A. B., Mendonca, P. O. R. de, Fortes, M. A. S., Tomaz, I., Pecorali, V. L., Auricino, T. B., et al. (2017). SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance. Journal of Endocrinology, 235( 3), 207-222. doi:10.1530/JOE-17-0255
    • NLM

      Pedroso JAB, Mendonca POR de, Fortes MAS, Tomaz I, Pecorali VL, Auricino TB, Costa IC, Lima LB, Furigo IC, Bueno DN, Ramos-Lobo AM, Lotfi CFP, Donato Junior J. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance [Internet]. Journal of Endocrinology. 2017 ; 235( 3): 207-222.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-17-0255
    • Vancouver

      Pedroso JAB, Mendonca POR de, Fortes MAS, Tomaz I, Pecorali VL, Auricino TB, Costa IC, Lima LB, Furigo IC, Bueno DN, Ramos-Lobo AM, Lotfi CFP, Donato Junior J. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance [Internet]. Journal of Endocrinology. 2017 ; 235( 3): 207-222.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-17-0255
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      AMARAL, Maria E. C. et al. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy. Journal of Endocrinology, v. 183, p. 469-476, 2004Tradução . . Disponível em: https://doi.org/10.1677/joe.1.05547. Acesso em: 22 maio 2024.
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      Amaral, M. E. C., Cunha, D. A., Anhê, G. F., Ueno, M., Carneiro, E. M., Velloso, L. A., et al. (2004). Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy. Journal of Endocrinology, 183, 469-476. doi:10.1677/joe.1.05547
    • NLM

      Amaral MEC, Cunha DA, Anhê GF, Ueno M, Carneiro EM, Velloso LA, Bordin S, Boschero AC. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy [Internet]. Journal of Endocrinology. 2004 ; 183 469-476.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.05547
    • Vancouver

      Amaral MEC, Cunha DA, Anhê GF, Ueno M, Carneiro EM, Velloso LA, Bordin S, Boschero AC. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy [Internet]. Journal of Endocrinology. 2004 ; 183 469-476.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.05547
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      OTTON, Rosemari et al. Non-esterified fatty acids and human lymphocyte death: a mechanism that involves calcium release and oxidative stress. Journal of Endocrinology, v. 195, n. 1, p. 133-143, 2007Tradução . . Disponível em: https://doi.org/10.1530/joe-07-0195e. Acesso em: 22 maio 2024.
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      Otton, R., Silva, D. O. da, Campoio, T. R., Silveira, L. R., Souza, M. O. de, Hatanaka, E., & Curi, R. (2007). Non-esterified fatty acids and human lymphocyte death: a mechanism that involves calcium release and oxidative stress. Journal of Endocrinology, 195( 1), 133-143. doi:10.1530/joe-07-0195e
    • NLM

      Otton R, Silva DO da, Campoio TR, Silveira LR, Souza MO de, Hatanaka E, Curi R. Non-esterified fatty acids and human lymphocyte death: a mechanism that involves calcium release and oxidative stress [Internet]. Journal of Endocrinology. 2007 ; 195( 1): 133-143.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/joe-07-0195e
    • Vancouver

      Otton R, Silva DO da, Campoio TR, Silveira LR, Souza MO de, Hatanaka E, Curi R. Non-esterified fatty acids and human lymphocyte death: a mechanism that involves calcium release and oxidative stress [Internet]. Journal of Endocrinology. 2007 ; 195( 1): 133-143.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/joe-07-0195e
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      TAKADA, Julie et al. Metabolic recovery of adipose tissue is associated with improvement in insulin resistance in a model of experimental diabetes. Journal of Endocrinology, v. 198, n. 1, p. 51-60, 2008Tradução . . Disponível em: https://doi.org/10.1677/joe-08-0072. Acesso em: 22 maio 2024.
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      Takada, J., Fonseca-Alaniz, M. H., Campos, T. B. F. de, Andreotti, S., Campana, A. B., Okamoto, M. M., et al. (2008). Metabolic recovery of adipose tissue is associated with improvement in insulin resistance in a model of experimental diabetes. Journal of Endocrinology, 198( 1), 51-60. doi:10.1677/joe-08-0072
    • NLM

      Takada J, Fonseca-Alaniz MH, Campos TBF de, Andreotti S, Campana AB, Okamoto MM, Borges-Silva C das N, Machado UF, Lima FB. Metabolic recovery of adipose tissue is associated with improvement in insulin resistance in a model of experimental diabetes [Internet]. Journal of Endocrinology. 2008 ; 198( 1): 51-60.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe-08-0072
    • Vancouver

      Takada J, Fonseca-Alaniz MH, Campos TBF de, Andreotti S, Campana AB, Okamoto MM, Borges-Silva C das N, Machado UF, Lima FB. Metabolic recovery of adipose tissue is associated with improvement in insulin resistance in a model of experimental diabetes [Internet]. Journal of Endocrinology. 2008 ; 198( 1): 51-60.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe-08-0072
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SIMÕES, Daniel et al. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, v. 231, n. 3, p. 235-244, 2016Tradução . . Disponível em: https://doi.org/10.1530/JOE-16-0259. Acesso em: 22 maio 2024.
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      Simões, D., Riva, P., Peliciari-Garcia, R. A., Cruzat, V. F., Graciano, M. fernanda, Munhoz, A. C., et al. (2016). Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, 231( 3), 235-244. doi:10.1530/JOE-16-0259
    • NLM

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-16-0259
    • Vancouver

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-16-0259
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      CEDDIA, R. B. et al. Leptin inhibits insulin-stimulated incorporation of glucose into lipids and stimulates glucose decarboxylation in isolated rat adipocytes. Journal of Endocrinology, v. 158, p. R7-R9, 1998Tradução . . Disponível em: https://doi.org/10.1677/joe.0.158r007. Acesso em: 22 maio 2024.
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      Ceddia, R. B., William Júnior, W. N., Lima, F. B., & Curi, R. (1998). Leptin inhibits insulin-stimulated incorporation of glucose into lipids and stimulates glucose decarboxylation in isolated rat adipocytes. Journal of Endocrinology, 158, R7-R9. doi:10.1677/joe.0.158r007
    • NLM

      Ceddia RB, William Júnior WN, Lima FB, Curi R. Leptin inhibits insulin-stimulated incorporation of glucose into lipids and stimulates glucose decarboxylation in isolated rat adipocytes [Internet]. Journal of Endocrinology. 1998 ; 158 R7-R9.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.158r007
    • Vancouver

      Ceddia RB, William Júnior WN, Lima FB, Curi R. Leptin inhibits insulin-stimulated incorporation of glucose into lipids and stimulates glucose decarboxylation in isolated rat adipocytes [Internet]. Journal of Endocrinology. 1998 ; 158 R7-R9.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.158r007
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      WILLIAN, W N. e CEDDIA, R B. e CURI, Rui. Leptin controls the fate of fatty acids in isolated rat white adipocytes. Journal of Endocrinology, v. 175, p. 735-744, 2002Tradução . . Acesso em: 22 maio 2024.
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      Willian, W. N., Ceddia, R. B., & Curi, R. (2002). Leptin controls the fate of fatty acids in isolated rat white adipocytes. Journal of Endocrinology, 175, 735-744.
    • NLM

      Willian WN, Ceddia RB, Curi R. Leptin controls the fate of fatty acids in isolated rat white adipocytes. Journal of Endocrinology. 2002 ;175 735-744.[citado 2024 maio 22 ]
    • Vancouver

      Willian WN, Ceddia RB, Curi R. Leptin controls the fate of fatty acids in isolated rat white adipocytes. Journal of Endocrinology. 2002 ;175 735-744.[citado 2024 maio 22 ]
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BARBOSA, Helena C. et al. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway. Journal of Endocrinology, v. 199, n. 2, p. 299-306, 2008Tradução . . Disponível em: https://doi.org/10.1677/joe-08-0309. Acesso em: 22 maio 2024.
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      Barbosa, H. C., Bordin, S., Anhê, G. F., Persaud, S. J., Bowe, J., Borelli, M. I., et al. (2008). Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway. Journal of Endocrinology, 199( 2), 299-306. doi:10.1677/joe-08-0309
    • NLM

      Barbosa HC, Bordin S, Anhê GF, Persaud SJ, Bowe J, Borelli MI, Gagliardino JJ, Boschero AC. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway [Internet]. Journal of Endocrinology. 2008 ; 199( 2): 299-306.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe-08-0309
    • Vancouver

      Barbosa HC, Bordin S, Anhê GF, Persaud SJ, Bowe J, Borelli MI, Gagliardino JJ, Boschero AC. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway [Internet]. Journal of Endocrinology. 2008 ; 199( 2): 299-306.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe-08-0309
  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, ANATOMIA, CONEXINAS, NEURÔNIOS, OBESIDADE, CAMUNDONGOS, DIETA ANIMAL, HIPOTÁLAMO, JEJUNO DE ANIMAL, HOMEOSTASE

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      SOUZA, Gabriel Orefice de et al. Gap junctions regulate the activity of AgRP neurons and diet-induced obesity in male mice. Journal of Endocrinology, v. 255, n. 2, p. 75-90, 2022Tradução . . Disponível em: https://doi.org/10.1530/JOE-22-0012. Acesso em: 22 maio 2024.
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      Souza, G. O. de, Chaves, F. M., Silva, J. do N., Pedroso, J. A. B., Metzger, M. A., Frazão, R., & Donato Junior, J. (2022). Gap junctions regulate the activity of AgRP neurons and diet-induced obesity in male mice. Journal of Endocrinology, 255( 2), 75-90. doi:10.1530/JOE-22-0012
    • NLM

      Souza GO de, Chaves FM, Silva J do N, Pedroso JAB, Metzger MA, Frazão R, Donato Junior J. Gap junctions regulate the activity of AgRP neurons and diet-induced obesity in male mice [Internet]. Journal of Endocrinology. 2022 ; 255( 2): 75-90.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0012
    • Vancouver

      Souza GO de, Chaves FM, Silva J do N, Pedroso JAB, Metzger MA, Frazão R, Donato Junior J. Gap junctions regulate the activity of AgRP neurons and diet-induced obesity in male mice [Internet]. Journal of Endocrinology. 2022 ; 255( 2): 75-90.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-22-0012
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      COSTA ROSA, Luís Fernando Bicudo Pereira e CURY, Y e CURI, Rui. Effects of insulin , glucocorticoids and thyroid hormones on the activities of key enzymes of glycolysis, glutaminolysis, the pentose-phosphate pathway and the krebs cycle in rat macrophages. Journal of Endocrinology, v. 135, p. 213-9, 1992Tradução . . Disponível em: https://doi.org/10.1677/joe.0.1350213. Acesso em: 22 maio 2024.
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      Costa Rosa, L. F. B. P., Cury, Y., & Curi, R. (1992). Effects of insulin , glucocorticoids and thyroid hormones on the activities of key enzymes of glycolysis, glutaminolysis, the pentose-phosphate pathway and the krebs cycle in rat macrophages. Journal of Endocrinology, 135, 213-9. doi:10.1677/joe.0.1350213
    • NLM

      Costa Rosa LFBP, Cury Y, Curi R. Effects of insulin , glucocorticoids and thyroid hormones on the activities of key enzymes of glycolysis, glutaminolysis, the pentose-phosphate pathway and the krebs cycle in rat macrophages [Internet]. Journal of Endocrinology. 1992 ;135 213-9.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1350213
    • Vancouver

      Costa Rosa LFBP, Cury Y, Curi R. Effects of insulin , glucocorticoids and thyroid hormones on the activities of key enzymes of glycolysis, glutaminolysis, the pentose-phosphate pathway and the krebs cycle in rat macrophages [Internet]. Journal of Endocrinology. 1992 ;135 213-9.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1350213
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      OTTON, Rosemari et al. Diabetes induces apoptosis in lymphocytes. Journal of Endocrinology, v. 182, p. 145-156, 2004Tradução . . Disponível em: https://doi.org/10.1677/joe.0.1820145. Acesso em: 22 maio 2024.
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      Otton, R., Soriano, F. G., Verlengia Rozangela,, & Curi, R. (2004). Diabetes induces apoptosis in lymphocytes. Journal of Endocrinology, 182, 145-156. doi:10.1677/joe.0.1820145
    • NLM

      Otton R, Soriano FG, Verlengia Rozangela, Curi R. Diabetes induces apoptosis in lymphocytes [Internet]. Journal of Endocrinology. 2004 ; 182 145-156.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1820145
    • Vancouver

      Otton R, Soriano FG, Verlengia Rozangela, Curi R. Diabetes induces apoptosis in lymphocytes [Internet]. Journal of Endocrinology. 2004 ; 182 145-156.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1820145
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      OTTON, Rosemari e MENDONÇA, J R. e CURI, Rui. Diabetes causes marked changes in lymphocyte metabolism. Journal of Endocrinology, v. 174, p. 55-61, 2002Tradução . . Disponível em: https://doi.org/10.1677/joe.0.1740055. Acesso em: 22 maio 2024.
    • APA

      Otton, R., Mendonça, J. R., & Curi, R. (2002). Diabetes causes marked changes in lymphocyte metabolism. Journal of Endocrinology, 174, 55-61. doi:10.1677/joe.0.1740055
    • NLM

      Otton R, Mendonça JR, Curi R. Diabetes causes marked changes in lymphocyte metabolism [Internet]. Journal of Endocrinology. 2002 ; 174 55-61.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1740055
    • Vancouver

      Otton R, Mendonça JR, Curi R. Diabetes causes marked changes in lymphocyte metabolism [Internet]. Journal of Endocrinology. 2002 ; 174 55-61.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.0.1740055
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      ALBA-LOUREIRO, Tatiana Carolina et al. Diabetes causes marked changes in function and metabolism of rat neutrophils. Journal of Endocrinology, v. 188, n. 2, p. 295-303, 2006Tradução . . Disponível em: https://doi.org/10.1677/joe.1.06438. Acesso em: 22 maio 2024.
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      Alba-Loureiro, T. C., Hirabara, S. M., Mendonça, J. R., Curi, R., & Pithon-Curi, T. C. (2006). Diabetes causes marked changes in function and metabolism of rat neutrophils. Journal of Endocrinology, 188( 2), 295-303. doi:10.1677/joe.1.06438
    • NLM

      Alba-Loureiro TC, Hirabara SM, Mendonça JR, Curi R, Pithon-Curi TC. Diabetes causes marked changes in function and metabolism of rat neutrophils [Internet]. Journal of Endocrinology. 2006 ; 188( 2): 295-303.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.06438
    • Vancouver

      Alba-Loureiro TC, Hirabara SM, Mendonça JR, Curi R, Pithon-Curi TC. Diabetes causes marked changes in function and metabolism of rat neutrophils [Internet]. Journal of Endocrinology. 2006 ; 188( 2): 295-303.[citado 2024 maio 22 ] Available from: https://doi.org/10.1677/joe.1.06438
  • Source: Journal of Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA, BIOFISICA, INGESTÃO, HIPOTÁLAMO, OBESIDADE, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO, BALANÇO DE ENERGIA, JEJUNO DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, PESOS E MEDIDAS CORPORAIS

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      TEIXEIRA, Pryscilla D. S. et al. Characterization of the onset of leptin effects on the regulation of energy balance. Journal of Endocrinology, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.1530/JOE-20-0076. Acesso em: 22 maio 2024.
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      Teixeira, P. D. S., Ramos-Lobo, A. M., Tavares, M. R., Wasinski, F., Frazão, R., & Donato Junior, J. (2021). Characterization of the onset of leptin effects on the regulation of energy balance. Journal of Endocrinology, 1-13. doi:10.1530/JOE-20-0076
    • NLM

      Teixeira PDS, Ramos-Lobo AM, Tavares MR, Wasinski F, Frazão R, Donato Junior J. Characterization of the onset of leptin effects on the regulation of energy balance [Internet]. Journal of Endocrinology. 2021 ; 1-13.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-20-0076
    • Vancouver

      Teixeira PDS, Ramos-Lobo AM, Tavares MR, Wasinski F, Frazão R, Donato Junior J. Characterization of the onset of leptin effects on the regulation of energy balance [Internet]. Journal of Endocrinology. 2021 ; 1-13.[citado 2024 maio 22 ] Available from: https://doi.org/10.1530/JOE-20-0076

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